Friday, September 14, 2012

Sweden finds the heart can control fatty acid to take in and help and study the diabetic treatment

Sweden finds the heart can control fatty acid to take in and help and study the diabetic treatment
The intersection of forest and the intersection of Sri Lankan and card, Carol of Swedish, medical college announce 15, this organization expert find heart blood vessel and muscle can regulate, take in fatty acid in the the food while being daily while being spontaneous, this is significant for metabolism disease including 2 -type diabetes of treatment.
The researchers of the card medical college of Carol's forest Sri Lankan have carried on the experiment with the mouse, they conduct V E G F - B protein as being from muscle to blood vessel wall the signal, find its biological function after being paid and tests, this protein can control fatty acid in the blood vessel wall to transport protein (F a T P s) Quantity.
Swedish expert points out, when F T P s protein increases, the fat crossed and taken in from the blood vessel wall runs up correspondingly. Lack the mouse of V E G F - B protein receptor in wall of those blood vessels, its fat taken in in the muscle and heart is less, fat gathered in different organisms are organized is fewer too.
Person in charge of research project, medical biochemistry and biophysics professor Ulf and Eriksson of the card medical college of forest Sri Lankan of Carol say, then the important reason which suffers from the 2 -type diabetes that the excessive increase of fat because of muscles causes the human body to present insulin to resist, find, there are important meanings for the intersection of treatment and including 2 -type diabetes the intersection of metabolism and disease while being above-mentioned. This research results have already been issued in newest Britain>On the magazine.
According to the introduction of Eriksson,et al., they will be devoted to the next step to study how to influence the insulinogenic signal, and reduce the blood glucose level of suffering from the diabetic mouse by regulating V E G F - B protein signal. (Reporter Sun Qi )
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